Initial Therapeutic Focus
Addressing Muscle Wasting in the ICU
- Prolonged MV leads to Ventilator Induced Lung Injury (VILI) and Ventilation Induced Diaphragmatic Dysfunction (VIDD), resulting in delayed and complicated weaning process, which is associated with higher mortality and morbidity rates1
~30% of the general ICU population develops Critical Illness Myopathy (CIM), a common neuro-muscular complication of ICU treatment
- 100% of patients exposed to long-term controlled mechanical ventilation develop CIM
The cost of weaning ICU patients from MV exceeds $100 billion a year (mgt. est.) in the U.S. and Europe.
MV usage is also associated with other iatrogenic complications, including ventilator-associated brain injury (VABI).2
- It can cause secondary brain injury in patients with established primary brain injury (i.e., stroke or traumatic brain injury) and can also be a primary cause of brain injury in patients without antecedent brain injury.
1/ MV typically results in more severe impact in older patients due to structural age-related changes in lung tissue. See here.
2/ For more information regarding VABI, readers may reference: Ventilator-associated Brain Injury: A New Priority for Research in Mechanical Ventilation.
Leksum believes the standard of care in ICUs will be for all patients who are anticipated to be mechanically ventilated for an extended period (i.e., more than a day or two) to be considered candidates to receive Leksum’s LK-ICU Protocol, which is currently defined as the administration of multiple doses of LK-ICU beginning the first day patients are put on mechanical ventilation and continuing every other day until no longer needed. This prophylactic intervention is expected to reduce (a) VILI, (b) muscle dysfunction including CIM and VIDD, both of which can result from VILI, and also (c) the risk of VABI. Our current hypothesis is that the lung injury induced by MV induces the release of factors which negatively affect muscle and brain function.